Saturday, October 25, 2003
THE NEW YORK TIMES GETS YET ANOTHER
EPIDEMIC WRONG
The New York Times, which to this day still has not reported the true story about AIDS, CFIDS and HHV-6, now has shifted its monumental journalistic incompetence to the subject of SARS. An editorial in the Sunday paper gets it exactly wrong: "Health authorities are preparing for the possible return of the SARS virus this fall, but with any luck they should be able to prevent a global epidemic. The virus is a nasty germ that can inflict terrible harm on anyone who contracts SARS. But the virus is relatively hard to spread and the conditions that allowed the disease to race from nation to nation last season seem unlikely to repeat themselves."
Hard to spread? They should pay an intern to just Google the story. If they did they would know that many more people are positive for exposure to the virus than got sick from it. Many people who handled wild animals in Guangdong Province in China have evidence of exposure to the virus--as many as 40% in one market.
The Times should consider sending a reporter to interview Henry Niman, the Harvard professor who has watched the Sars epidemic like a hawk for much of the last year. Niman has predicted for months that when the weather turns colder in China and elsewhere, a SARS apocalypse could occur. Since many animals are capable of contracting the virus, there could be many new reservoirs when the next SARS season begins.
The Times editorial comes close to chiding the CDC for panic-mongering. The Times should really try to get to the bottom of the SARS story before they make a judgment like that. They may have to eat their words. They should try and ascertain whether the CDC knows more than it is telling the public about the likelihood of an explosive new SARS epidemic. If Henry Niman's vision of the future is correct, we could have a major catastrophe in the United States and elsewhere this winter. SARS could end up being the number one issue in the next election.
One more suggestion for the Times. Investigate the role of pigs in the SARS epidemic. We predict they'll find a big story there.
EPIDEMIC WRONG
The New York Times, which to this day still has not reported the true story about AIDS, CFIDS and HHV-6, now has shifted its monumental journalistic incompetence to the subject of SARS. An editorial in the Sunday paper gets it exactly wrong: "Health authorities are preparing for the possible return of the SARS virus this fall, but with any luck they should be able to prevent a global epidemic. The virus is a nasty germ that can inflict terrible harm on anyone who contracts SARS. But the virus is relatively hard to spread and the conditions that allowed the disease to race from nation to nation last season seem unlikely to repeat themselves."
Hard to spread? They should pay an intern to just Google the story. If they did they would know that many more people are positive for exposure to the virus than got sick from it. Many people who handled wild animals in Guangdong Province in China have evidence of exposure to the virus--as many as 40% in one market.
The Times should consider sending a reporter to interview Henry Niman, the Harvard professor who has watched the Sars epidemic like a hawk for much of the last year. Niman has predicted for months that when the weather turns colder in China and elsewhere, a SARS apocalypse could occur. Since many animals are capable of contracting the virus, there could be many new reservoirs when the next SARS season begins.
The Times editorial comes close to chiding the CDC for panic-mongering. The Times should really try to get to the bottom of the SARS story before they make a judgment like that. They may have to eat their words. They should try and ascertain whether the CDC knows more than it is telling the public about the likelihood of an explosive new SARS epidemic. If Henry Niman's vision of the future is correct, we could have a major catastrophe in the United States and elsewhere this winter. SARS could end up being the number one issue in the next election.
One more suggestion for the Times. Investigate the role of pigs in the SARS epidemic. We predict they'll find a big story there.
Wednesday, October 22, 2003
NEW RESEARCH BOLSTERS CLAIMS THAT HHV-6
PLAYS A MAJOR ROLE IN AIDS
Is HHV-6 the real AIDS virus?
New research published in Blood on October 15 by a team of researchers that includes famous HHV-6 researcher Paolo Lusso, suggests that HHV-6 can play a major role in suppressing the immune system.
The abstract of the research follows:
"Human herpesvirus 6 (HHV-6) is a potentially immunosuppressive agent that has been suggested to act as a cofactor in the progression of HIV disease. Exposure of human macrophages to HHV-6A or HHV-6B profoundly impaired their ability to produce interleukin 12 (IL-12) upon stimulation with interferon-gamma (IFN-gamma) and lipopolysaccharide (LPS). By contrast, the production of tumor necrosis factor-alpha (TNF-alpha); regulated on activation, normal T-cell expressed and secreted (RANTES); and macrophage inflammatory protein 1beta (MIP-1beta) was not negatively affected. To exclude the involvement of IL-12-suppressive cytokines, such as IL-10 and TNF-alpha, the viral stocks were fractionated by ultra-centrifugation. The bulk of the suppressive activity was recovered within the virion-rich pelleted fraction that was virtually devoid of such cytokines. IL-12 suppression was independent of viral replication, and the effect was not abrogated upon ultraviolet-light inactivation of the viral inoculum. The mechanism of HHV-6-mediated IL-12 suppression was investigated by RNase protection assays, which demonstrated unaltered levels of IL-12 p35 mRNA and only a modest reduction in p40 mRNA, which was insufficient to account for the near-complete loss of both extracellular and intracellular IL-12 protein. Moreover, both the IFN-gamma and the LPS signaling pathways were intact in HHV-6-treated cells. These data suggest that HHV-6 can dramatically affect the generation of effective cellular immune responses, providing a novel potential mechanism of HHV-6-mediated immunosuppression."
It is truly tragic that the HIV Mafia continues dismiss any suggestion that HHV-6 needs to be treated in AIDS. Not only do we think it needs to be treated, but we also think it is the real AIDS virus and that anyone who has died of AIDS since HHV-6 was discovered in 1986 was a victim of medical politics and scientific fraud. If HHV-6 had been recognized as the real AIDS killer, these victims could have been treated with much more effective medications. The only reason HHV-6 is not the subject to multi-million dollar research is that the HIV establishment is afraid their cockamamie HIV paradigm will be seen for the outrageous lie that it is. Hopefully, one of these days a massive--and we mean massive--class action lawsuit will be launched against the CDC, the NIH and all of the scientific con artists who have perpetrated this tragic HIV fraud. And the suit should also be done in the name of CFIDS patients, MS patients and all the other patients who are suffering from HHV-6 immunological damage.
PLAYS A MAJOR ROLE IN AIDS
Is HHV-6 the real AIDS virus?
New research published in Blood on October 15 by a team of researchers that includes famous HHV-6 researcher Paolo Lusso, suggests that HHV-6 can play a major role in suppressing the immune system.
The abstract of the research follows:
"Human herpesvirus 6 (HHV-6) is a potentially immunosuppressive agent that has been suggested to act as a cofactor in the progression of HIV disease. Exposure of human macrophages to HHV-6A or HHV-6B profoundly impaired their ability to produce interleukin 12 (IL-12) upon stimulation with interferon-gamma (IFN-gamma) and lipopolysaccharide (LPS). By contrast, the production of tumor necrosis factor-alpha (TNF-alpha); regulated on activation, normal T-cell expressed and secreted (RANTES); and macrophage inflammatory protein 1beta (MIP-1beta) was not negatively affected. To exclude the involvement of IL-12-suppressive cytokines, such as IL-10 and TNF-alpha, the viral stocks were fractionated by ultra-centrifugation. The bulk of the suppressive activity was recovered within the virion-rich pelleted fraction that was virtually devoid of such cytokines. IL-12 suppression was independent of viral replication, and the effect was not abrogated upon ultraviolet-light inactivation of the viral inoculum. The mechanism of HHV-6-mediated IL-12 suppression was investigated by RNase protection assays, which demonstrated unaltered levels of IL-12 p35 mRNA and only a modest reduction in p40 mRNA, which was insufficient to account for the near-complete loss of both extracellular and intracellular IL-12 protein. Moreover, both the IFN-gamma and the LPS signaling pathways were intact in HHV-6-treated cells. These data suggest that HHV-6 can dramatically affect the generation of effective cellular immune responses, providing a novel potential mechanism of HHV-6-mediated immunosuppression."
It is truly tragic that the HIV Mafia continues dismiss any suggestion that HHV-6 needs to be treated in AIDS. Not only do we think it needs to be treated, but we also think it is the real AIDS virus and that anyone who has died of AIDS since HHV-6 was discovered in 1986 was a victim of medical politics and scientific fraud. If HHV-6 had been recognized as the real AIDS killer, these victims could have been treated with much more effective medications. The only reason HHV-6 is not the subject to multi-million dollar research is that the HIV establishment is afraid their cockamamie HIV paradigm will be seen for the outrageous lie that it is. Hopefully, one of these days a massive--and we mean massive--class action lawsuit will be launched against the CDC, the NIH and all of the scientific con artists who have perpetrated this tragic HIV fraud. And the suit should also be done in the name of CFIDS patients, MS patients and all the other patients who are suffering from HHV-6 immunological damage.
